Benfotiamine for Diabetes and Blood Sugar Support

If you have high blood sugar, you worry about your eyes, your kidneys, and your feet. But what about your brain?

High glucose levels are notoriously toxic to nerve tissue. Over time, sugar "gums up" the microscopic blood vessels in your brain and nerves, leading to cognitive decline and painful neuropathy. This is where Benfotiamine shines.

Benfotiamine is a fat-soluble form of Vitamin B1 that has become a staple in diabetic support protocols in Europe for decades. Here is why it is critical for blood sugar management.

The Mechanism: Stopping Glycation (AGEs)

When there is too much sugar in your blood, it binds to proteins and fats, creating sticky, damaging molecules called Advanced Glycation End-products (AGEs). Think of AGEs as "biological rust." They stiffen blood vessels and kill nerve endings.

Benfotiamine blocks this process. It activates an enzyme called Transketolase. This enzyme acts like a traffic cop, diverting excess sugar away from the pathways that create AGEs and into safe energy production pathways.

The Mechanism: B1 Deficiency in Diabetes

Diabetics are notoriously deficient in Vitamin B1 (Thiamine). High blood sugar causes the kidneys to flush Thiamine out of the body at a rapid rate.

Without Thiamine, your cells cannot process sugar into energy. This leads to profound fatigue. Taking standard water-soluble Thiamine often isn't enough because it is poorly absorbed. Benfotiamine is absorbed 3.6 times better, ensuring your tissues get the B1 they are losing.

The Protocol: Metabolic Defense

If you are managing blood sugar, you need more than just insulin control; you need tissue protection. The FOG OFF protocol is designed to defend against glucose toxicity.

1. The Anti-Glycation Agent: Benfotiamine

Included to protect nerves and brain cells from sugar damage.

• Mechanism: Increases Transketolase activity by 300-400%, effectively detoxifying glucose metabolites before they cause harm.

2. The Insulin Mimic: Alpha-Lipoic Acid

Alpha-Lipoic Acid (ALA) is arguably the most important supplement for diabetics alongside Benfotiamine.

• Mechanism: It improves insulin sensitivity (helping cells take in sugar) and neutralizes the free radicals caused by high glucose. In Germany, ALA is a prescription treatment for diabetic neuropathy.

Summary

You can't always control your blood sugar perfectly, but you can control how much damage it does. By using Benfotiamine to block glycation and ALA to improve sensitivity, you build a shield around your nervous system.

FOG OFF is your metabolic defense system.